Talk:VLA-4

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Wiki Education Foundation-supported course assignment[edit]

This article is or was the subject of a Wiki Education Foundation-supported course assignment. Further details are available on the course page. Student editor(s): Immcarle66.

Above undated message substituted from Template:Dashboard.wikiedu.org assignment by PrimeBOT (talk) 12:12, 17 January 2022 (UTC)[reply]

Introduction[edit]

Good afternoon ImmCarle66,

My name is ImmCarle75, and I look forward to seeing your updates and revisions.

Immcarle75 (talk) 15:26, 8 January 2018 (UTC)ImmCarle75[reply]

Hello! I am going to be trying to update this VLA-4 page as a part of an assignment for an immunology class. here are five sources I am going to be investigating in order to improve on this article.

Schmid, Michael C. et al. “PI3-Kinase Γ Promotes Rap1a-Mediated Activation of Myeloid Cell Integrin α4β1, Leading to Tumor Inflammation and Growth.” Ed. Donald Gullberg. PLoS ONE 8.4 (2013): e60226. PMC. Web. 31 Jan. 2018.

Garmy-Susini, Barbara et al. “PI3Kα Activates Integrin α4β1 to Establish a Metastatic Niche in Lymph Nodes.” Proceedings of the National Academy of Sciences of the United States of America 110.22 (2013): 9042–9047. PMC. Web. 31 Jan. 2018.

Lu, Ling et al. “Kindlin-3 Is Essential for the Resting α4β1 Integrin-Mediated Firm Cell Adhesion under Shear Flow Conditions.” The Journal of Biological Chemistry 291.19 (2016): 10363–10371. PMC. Web. 31 Jan. 2018.

Xu, Ying-zi, et al. “Arylsulfonamide Pyrimidines as VLA-4 Antagonists.” Bioorganic & Medicinal Chemistry Letters, vol. 23, no. 10, 2013, pp. 3070–3074.

Brachtl, Gabriele, et al. “Differential Bone Marrow Homing Capacity of VLA-4 and CD38 High Expressing Chronic Lymphocytic Leukemia Cells (Homing of High-Risk CLL Cells).” PLoS ONE, vol. 6, no. 8, 2011, p. e23758.

Immcarle66 (talk) 02:22, 31 January 2018 (UTC)[reply]

Alternate name?[edit]

I believe this is also called CDw49d in some literature — Preceding unsigned comment added by 129.241.229.131 (talk) 10:42, 4 September 2019 (UTC)[reply]