User:Strudel3/Mechanism of action of aspirin

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Article Draft[edit]

Action Mechanism of Aspirin Draft.[edit]

Aspirin, a non-steroidal anti-inflammatory drug (NSAIDs), is a commonly known blood thinner and anti-inflammatory. It functions by targeting enzymes involved in the signal pathways results in swelling and inflammation. ^[1] The molecule targets an enzyme which produces a conformational change in the chemical structure stopping the production of prostaglandins, which are located at the cite of abrasion and are associated with inflammation^[2]. The conformational change is absolute, effecting the cyclooxyrgenase (COX) class of enzymes more specifically COX-1 and COX-1, which are associated with the response to injury and infection.^[1]

Inhibition of Cyclooxyrgenase (COX)[edit]

Aspirin competes for the binding site on the COX enzyme, preventing other signal molecules from attaching and activating the enzyme. Two known COX enzyme subtypes are known targets of aspirin, COX-1 and COX-2

References[edit]

^ ^a ^b "How aspirin works". www.uchicagomedicine.org. Retrieved 2023-09-29.
^ Ricciotti, Emanuela; FitzGerald, Garret A. (2011-5). "Prostaglandins and Inflammation". Arteriosclerosis, thrombosis, and vascular biology. 31 (5): 986–1000. doi:10.1161/ATVBAHA.110.207449. ISSN 1079-5642. PMC 3081099. PMID 21508345. {{cite journal}}: Check date values in: |date= (help)

[:0-1] "How aspirin works". www.uchicagomedicine.org. Retrieved 2023-09-29.

[2] Ricciotti, Emanuela; FitzGerald, Garret A. (2011-5). "Prostaglandins and Inflammation". Arteriosclerosis, thrombosis, and vascular biology. 31 (5): 986–1000. doi:10.1161/ATVBAHA.110.207449. ISSN 1079-5642. PMC 3081099. PMID 21508345. {{cite journal}}: Check date values in: |date= (help)

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