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Welcome[edit]

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– the WikiProject Medicine team Doc James (talk · contribs · email) 22:16, 31 January 2020 (UTC)[reply]

Acute bronchitis[edit]

How does this related

"Resistance to antibiotics may develop rapidly and last for 10 to 14 days. Thus, patients who are taking amoxicillin for acute bronchitis should be prescribed another type of antibiotic, (such as clindamycin or a cephalosporin) when an antibiotic is needed for an odontogenic infection. [1]"


Why would people be taking amoxicillin for acute bronchitis, a viral infection? And viruses do not develop resistance... Doc James (talk · contribs · email) 22:18, 31 January 2020 (UTC)[reply]

Text[edit]

"== Oral Manifestations and Complications == Nasal symptoms, allergic rhinitis, and mouth breathing are common with extrinsic asthma. Patients with asthma who are mouth breathers may have altered naso-respira­tory function, which may be associated with increased upper anterior and total anterior facial height, higher palatal vault, greater overjet, and a higher prevalence of crossbite.[2] The medications taken by patients who have asthma may contribute to oral disease. For example, β2-agonist inhalers reduce salivary flow by 20% to 35%, decrease plaque pH, and are associated with increased preva­lence of gingivitis and caries in patients with moderate to severe asthma.[3] Gastroesophageal acid reflux is common in patients with asthma and is exacerbated by the use of β-agonists and theophylline. This reflux can contribute to erosion of enamel. Oral candidiasis (acute pseudomembranous type) occurs in approximately 5% of patients who use inhalation steroids for long periods at high dose or frequency. [4]However, development of this condition is rare if a “spacer” or aerosol-holding chamber is attached to the metered-dose inhaler and the mouth is rinsed with water after each use.[5] The condi­tion readily responds to local antifungal therapy (i.e., nystatin, clotrimazole, or fluconazole). Patients should receive instructions on the proper use of their inhaler and the need for oral rinsing. Headache is a frequent adverse effect of antileukotrienes and theophylline. The clinician should be aware of this adverse effect when diagnosing disease in patients with orofacial pain complaints." This needs work. Some of the refs are super old. Others are primary and others do not support the content in question. Doc James (talk · contribs · email) 23:32, 31 January 2020 (UTC)[reply]

References

  1. ^ Greenberg, Glick, Ship. Burket’s Oral Medicine 11th edition, Chapter 12: Diseases of Respiratory Tract,. pp. page 305-306. {{cite book}}: |pages= has extra text (help)CS1 maint: multiple names: authors list (link)
  2. ^ Bresolin D; et al. (1983). "Mouth breathing in allergic children: its relationship to dentofacial development". Am J Orthod. 83: 334–340. {{cite journal}}: Explicit use of et al. in: |last= (help)
  3. ^ Reddy DK, Hegde AM, Munshi AK (2003). "Dental caries status of children with bronchial asthma". J Clin Pediatr Dent. 27: 293–295.{{cite journal}}: CS1 maint: multiple names: authors list (link)
  4. ^ "Diagnostic standards and classification of tuberculosis in adults and children". Am J Respir Crit Care Med. 161(4 Pt 1): 1376–1395. 2000.
  5. ^ "Drugs for ambulatory asthma". Med Lett Drugs Ther. 33: 9–12. 1991.

This has similar issues[edit]

Also why does management have a capital? Much of this is unsupported... Doc James (talk · contribs · email) 23:35, 31 January 2020 (UTC)[reply]

Dental Management[edit]

The underlying primary goal in dental management of patients with asthma is to prevent an acute asthma attack. The first step in achieving this goal is to iden­tify patients with asthma by history, followed by assess­ment to elucidate the surrounding details of the problem, along with prevention of precipitating factors.

Through a good history, the dentist should be able to determine the severity and stability of disease. Questions should be asked that ascertain the type of asthma (e.g., allergic versus nonallergic), the precipitating substances, the frequency and severity of attacks, the times of day when attacks occur, whether this is a current or past problem, how attacks usually are managed, and whether the patient has received emergency treatment for an acute attack. The clinician must be cognizant of the indications of severe disease: frequent exacerbations, exercise intolerance, FEV1 less than 60%, use of several medications, and a history of visits to an emergency facility for treatment of acute attack.

The stability of the disease can be assessed during the interview component of the history and by clinical exam­ination and the results of laboratory measures. Features such as shortness of breath, wheezing, increased respira­tory rate (more than 50% above normal), FEV1 that has fallen more than 10% or to below 80% of peak FEV1, an eosinophil count that is elevated to above 50/mm3, poor drug use compliance, and emergency department visits within the previous 3 months suggest inadequate treatment and poor stability. Also, the use of more than 1.5 canisters of a beta agonist inhaler per month (more than 200 inhalations per month) or doubling of monthly use indicates high risk for a severe asthma attack.[1] For severe and unstable asthma, consultation with the patient’s physician is advised. Routine dental treatment should be postponed until better control is achieved.

Modifications during the preoperative and operative phases of dental management of a patient with asthma can minimize the likelihood of an attack. Patients who have nocturnal asthma should be scheduled for late-morning appointments, when attacks are less likely. Use of operatory odorants (e.g., methyl methacrylate) should be reduced before the patient is treated. Patients should be instructed to regularly use their medications, to bring their inhalers (bronchodilators) to each appointment, and to inform the dentist at the earliest sign or symptom of an asthma attack. Prophylactic inhalation of a patient’s bronchodilator at the beginning of the appointment is a valuable method of preventing an asthma attack. Alter­natively, patients may be advised to bring their spirom­eter and daily expiratory record to the office. The dentist may request that the patient exhale into the spirometer and record the expired volume. A significant drop in lung function (to below 80% of peak FEV1 or a greater than 10% drop from previously recorded values) indicates that prophylactic use of the inhaler or referral to a physi­cian is needed.[2] The use of a pulse oximeter also is valu­able for determining the patient’s oxygen saturation level. In healthy patients, this value remains between 97% and 100%, whereas a drop to 91% or below indicates poor oxygen exchange and the need for intervention.

Because stress is implicated as a precipitating factor in asthma attacks and dental treatment may result in decreased lung function, all dental staff members should make every effort to identify patients who are anxious and provide a stress-free environment through establishment of rapport and openness. [3]Preoperative and intraoperative sedation may be desirable. If sedation is required, nitrous oxide–oxygen inhalation is best. Nitrous oxide is not a respiratory depressant, nor is it an irritant to the tracheobronchial tree. Oral premedica­tion may be accomplished with small doses of a short-acting benzodiazepine. Reasonable alternatives with children are hydroxyzine (Vistaril), for its antihistamine and sedative properties, and ketamine, which causes bronchodilation. Barbiturates and narcotics, particularly meperidine, are histamine-releasing drugs that can provoke an attack. Outpatient general anesthesia gener­ally is contraindicated for patients with asthma.

Selection of local anesthetic may require adjustment. In 1987, the U.S. Food and Drug Administration (FDA) warned that drugs that contained sulfites were a cause of allergic-type reactions in susceptible individu­als.[4] Sulfite preservatives are found in local anesthetic solutions that contain epinephrine or levonordefrin, although the amount of sulfite in a local anesthetic car­tridge is less than the amount commonly found in an average serving of certain foods. Although rare, at least one case of an acute asthma attack precipitated by expo­sure to sulfites has been reported.[5] Thus, the use of local anesthetic without epinephrine or levonordefrin may be advisable for patients with moderate to severe disease. Because relevant data remain limited, the dentist should discuss with the patient any past responses to local anes­thetics and allergy to sulfites and should consult with the physician on this issue. As an alternative, local anes­thetics without a vasoconstrictor may be used in at-risk patients.

Patients with asthma who are medicated over the long term with systemic corticosteroids may require supple­mentation for major surgical procedures if their health is poor. However, long-term use of inhaled corticosteroids rarely causes adrenal suppression unless the daily dosage exceeds 1.5 mg of beclometha­sone dipropionate or its equivalent. Administration of aspirin-containing medication or other nonsteroidal anti-inflammatory drugs to patients with asthma is not advisable, because aspirin ingestion is associated with the precipitation of asthma attacks in a small percentage of patients. Likewise, barbiturates and narcotics are best not used, because they also may precipitate an asthma attack. Antihistamines have ben­eficial properties but should be used cautiously because of their drying effects. Patients who are taking theophyl­line preparations should not be given macrolide antibiot­ics (i.e., erythromycin and azithromycin) or ciprofloxacin hydrochloride, because these agents interact with the­ophylline to produce a potentially toxic blood level of theophylline. To prevent serious toxicity, the dentist should ask the patient who takes theophylline whether the dosage is being monitored on the basis of serum theophylline levels (recommended to be less than 10 μg/ mL). Approximately 3% of patients who take zileuton exhibit elevated alanine transaminase levels, reflecting liver dysfunction that may affect the metabolism of den­tally administered drugs.[6]

References

  1. ^ Suissa S, Ernst P (1997). "Albuterol in mild asthma". N Engl J Med. 336: 729.
  2. ^ Ulrik CS, Frederiksen J (1995). "Mortality and markers of risk of asthma death among 1,075 outpatients with asthma". Chest. 108: 10–15.
  3. ^ Mathew T; et al. (1998). "Effect of dental treatment on the lung func¬tion of children with asthma". J Am Dent Assoc. 129: 1120–1128. {{cite journal}}: Explicit use of et al. in: |last= (help)
  4. ^ "U.S. Department of Health and Human Services: Warning on prescription drugs containing sulfites". FDA Drug Bull. 17: 2–3. 1987.
  5. ^ Schwartz HJ; et al. (1989). "Metabisulfite sensitivity and local dental anesthesia". Ann Allergy. 62: 83–86. {{cite journal}}: Explicit use of et al. in: |last= (help)
  6. ^ Elnabtity MH, et al 99(7 Suppl):S1-S6, 1999. (1999). "Leukotriene modifiers in the management of asthma,". J Am Osteopath Assoc. 99(7 Suppl): S1–S6.{{cite journal}}: CS1 maint: multiple names: authors list (link) CS1 maint: numeric names: authors list (link)

Text[edit]

Nasal symptoms, allergic rhinitis, and mouth breathing are common with extrinsic asthma. Patients with asthma who are mouth breathers may have altered naso-respira­tory function, which may be associated with increased upper anterior and total anterior facial height, higher palatal vault, greater overjet, and a higher prevalence of crossbite.[1]

Why a 1983 primary source[1]? Did you read any of the above... Doc James (talk · contribs · email) 02:19, 24 February 2020 (UTC)[reply]

The medications taken by patients who have asthma may contribute to oral disease. For example, β2-agonist inhalers reduce salivary flow by 20% to 35%, decrease plaque pH, and are associated with increased preva­lence of gingivitis and caries in patients with moderate to severe asthma.[2]

This is another oldish primary source.[2]


Gastroesophageal acid reflux is common in patients with asthma and is exacerbated by the use of β-agonists and theophylline. This reflux can contribute to erosion of enamel. Oral candidiasis (acute pseudomembranous type) occurs in approximately 5% of patients who use inhalation steroids for long periods at high dose or frequency.[3]

Why a paper on tuberculosis in the asthma article?[3] We already discuss thrush in the article. Doc James (talk · contribs · email) 02:22, 24 February 2020 (UTC)[reply]

However, development of this condition is rare if a “spacer” or aerosol-holding chamber is attached to the metered-dose inhaler and the mouth is rinsed with water after each use.[4] The condi­tion readily responds to local antifungal therapy (i.e, nystatin, clotrimazole, or fluconazole). Patients should receive instructions on the proper use of their inhaler and the need for oral rinsing. Headache is a frequent adverse effect of antileukotrienes and theophylline.

Did you read the recommendation above. Doc James (talk · contribs · email) 02:19, 24 February 2020 (UTC)[reply]

References

  1. ^ Bresolin D, et al Am J Orthod 83:334- 340, 1983. (1983). "Mouth breathing in allergic children: its relationship to dentofacial development". Am J Orthod. 83: 334–340.{{cite journal}}: CS1 maint: multiple names: authors list (link) CS1 maint: numeric names: authors list (link)
  2. ^ Reddy DK, Hegde AM, Munshi AK (2003). "Dental caries status of children with bronchial asthma". J Clin Pediatr Dent. 27: 293–295.{{cite journal}}: CS1 maint: multiple names: authors list (link)
  3. ^ "Diagnostic standards and classification of tuberculosis in adults and children". Am J Respir Crit Care Med. 161(4 Pt 1): 1376–1395. 2000.
  4. ^ "Drugs for ambulatory asthma". Med Lett Drugs Ther. 33: 9–12. 1991.

Rinsing the mouth[edit]

Have added the bit on rinsing here[4]

Thrush was already mentioned. Doc James (talk · contribs · email) 02:34, 24 February 2020 (UTC)[reply]

Text[edit]

A higher prevalence of caries is noted among patients with asthma, particularly children, that may be related to the disease and/or its treatment.[1] Asthma may impact dental caries susceptibility either directly through biological changes, such as salivary composition, or indirectly as side effects of the medicines used to control the symptoms of asthma. For example, the prolonged use of beta 2 agonists has been associated with a decrease in salivary production.Cite error: A <ref> tag is missing the closing </ref> (see the help page).[2] Beta 2 agonists work to relax constricted smooth muscles that comprise the airway. This tranquilizing effect occasionally impacts smooth muscles outside of the airway, such as the lower esophageal sphincter which prevents gastroesophageal acid reflux. However, when relaxed under the influence of beta 2 agonists, the smooth muscle allows erosive gastric acid to enter the mouth. Repeated exposure to such acidic conditions poses a significant threat to the dentition and may result in erosion of primary teeth in children. Both aerosol and powdered forms of asthma medications have a pH less than the critical level of pH 5.5 will lead to enamel dissolution. [3]

Teeth[edit]

Cavities occur more often in people with asthma.[4] This may be related to the effect of beta 2 agonists decreasing saliva.[5] These medications may also increase the risk of dental erosions.[5]

Adjusting to this. The other refs are either primary or do not mention asthma. Doc James (talk · contribs · email) 21:19, 26 February 2020 (UTC)[reply]
Okay adjusted the text and put it here https://en.wikipedia.org/w/index.php?title=Asthma&diff=942790878&oldid=942787799&diffmode=source Doc James (talk · contribs · email) 21:21, 26 February 2020 (UTC)[reply]
  1. ^ Agostini BA, Collares KF, dos Santos Costa F, Correa MB, Demarco FF. The role of asthma in caries occurrence — meta-analysis and meta-regression. J Asthma. 2018 Aug 24:1–12. {{cite journal}}: Cite journal requires |journal= (help); Missing or empty |title= (help)CS1 maint: multiple names: authors list (link) CS1 maint: numeric names: authors list (link)
  2. ^ Arafa A, Aldahlawi S, Fathi A. Assessment of the oral health status of asthmatic children. Eur J Dent. 2017;10:357. {{cite journal}}: Cite journal requires |journal= (help); Missing or empty |title= (help)CS1 maint: multiple names: authors list (link) CS1 maint: numeric names: authors list (link)
  3. ^ Dawes C. What is the critical pH and why does a tooth dissolve in acid? J Can Dent Assoc. 2003; 69(11):722-724. {{cite journal}}: Cite journal requires |journal= (help); Missing or empty |title= (help)CS1 maint: multiple names: authors list (link) CS1 maint: numeric names: authors list (link)
  4. ^ Agostini, BA; Collares, KF; Costa, FDS; Correa, MB; Demarco, FF (August 2019). "The role of asthma in caries occurrence - meta-analysis and meta-regression". The Journal of asthma : official journal of the Association for the Care of Asthma. 56 (8): 841–852. doi:10.1080/02770903.2018.1493602. PMID 29972654.
  5. ^ a b Thomas, MS; Parolia, A; Kundabala, M; Vikram, M (June 2010). "Asthma and oral health: a review". Australian dental journal. 55 (2): 128–33. doi:10.1111/j.1834-7819.2010.01226.x. PMID 20604752.
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